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The renin-aldosterone link is mediated by angiotensin II, so coadministration of the angiotensin II receptor antagonist tends to reverse the potassium loss associated with these diuretics.

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Irbesartan blocks the vasoconstrictor and aldosterone-secreting consequences of angiotensin II by selectively binding on the AT1 angiotensin II receptor. You can find also an AT2 receptor in several tissues, but It's not at all involved in cardiovascular homeostasis.

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Nursing Moms: It's not identified no matter if irbesartan is excreted in human milk, but irbesartan or some metabolite of irbesartan is secreted at small focus from the milk of lactating rats.

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Angiotensin II would or else bind to the AT1 receptor, inducing vasoconstriction and aldosterone secretion, raising hypertension.

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Mainly because irbesartan will not inhibit ACE, it doesn't affect the response to bradykinin; whether this has scientific relevance isn't regarded.

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